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Reportedly, CRP inhibits nitric oxide production [5] and increases the expression of adhesion molecules in endothelial cells [6]

Prediction accuracy for each genotype is also reported

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doi: 10.1111/jth.14234 Summary Keywords trimethylamine N-oxide, atherosclerosis, cardiovascular disease, inflammation mechanism, clinical prognostic stratification, therapy Citation Yang S, Li X, Yang F, Zhao R, Pan X, Liang J, Tian L, Li X, Liu L, Xing Y and Wu M (2019) Gut Microbiota-Dependent Marker TMAO in Promoting Cardiovascular Disease: Inflammation Mechanism, Clinical Prognostic, and Potential as a Therapeutic Target