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In recent in vitro assays performed by our group [32], we showed that H 2 O 2 -induced oxidative stress triggered the binding of Prx2 and GPx to RBC membrane
doi: 10.1016/S0168-3659(97)00154-5
The magnitude of Chac1 downregulation exceeded that of canonical refeeding-responsive genes controlling hepatic glucose metabolism, such as the downregulation of G6pc or upregulation of Gck (Figure 6A), indicating robust transcriptional suppression of Chac1 during feeding
10.1111/nyas.13008 [DOI] [PMC free article] [PubMed] [Google Scholar] 370.Tsoi, J
