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10.22074/cellj.2021.7183 160 LiuY.MyojinT.LiK.KuritaA.SetoM.MotoyamaA.et al (2022)

Consistent with decreased fasting glucose levels and improved insulin tolerance observed under chow, and to a lesser extent under HFD conditions (Figure 1, H and I, and Figure 2, J and L), GSH depletion significantly downregulated gluconeogenic enzymes G6pc and Pck1 in chow-fed mice, with no substantial change under HFD (Figure 3, A and B)

This duality positions GSH as a context- and compartment-dependent determinant of cellular redox thresholds and raises several unresolved questions that will shape future directions in redox biology and cell death research

doi: 10.1016/j.amjhyper.2006.08.009