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In this disease, the failure to upregulate CHAC1 is detrimental
Utility of PET, CT, and EUS to identify pathologic responders in esophageal cancer
Under normal conditions, APAP is metabolized via conjugation with sulfate and glucuronide, leading to its excretion in the urine
This induces DNA damage and adenosine triphosphate (ATP) depletion, and triggers ataxia telangiectasia mutated/adenosine monophosphateactivated protein kinase and inhibition of mammalian target of rapamycin in cancer cells (67)
