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This disruption in signaling pathways is a critical factor in tumor formation (Yewale et al., 2013), particularly in lung cancer, where EGFR plays a vital role (Peifer et al., 2012)

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This suggests that D-HIS exposure may non-specifically decrease the cellular GSH concentration, so resulting in an underestimation of the increase in cellular GSH level potentially attainable with other counter-selection systems

doi: 10.3389/fendo.2024.1492610 Summary Keywords ferroptosis, bone microenvironment, bone and joint diseases, mechanisms, therapeutic prospects Citation Xiao R, Han Z, Jia P, Li P, Gong M, Cai Y, Pang L, Ye X and Jin S (2025) Ferroptosis and bone health: bridging the gap between mechanisms and therapy