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The authors found that although ISL was not a direct inhibitor, its metabolite 2, 4, 2, 4-tetrahydroxychalcone (THC), which was a product of ISL-treated cells, directly interfered with the Src signaling pathway and inhibited Src kinase activity
The difference is that the pharmaceutical company was required to prove its product works before selling it
Furthermore, in these experiments (though not in an earlier study [15]), acetoacetate reduced excitatory glutamatergic synaptic transmission at hippocampal CA1 neurons (Figure 1)
Acta Neuropathol
