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Leopold F, Stefano T, Laura O

This issue is particularly concerning and has become a challenge for researchers aiming to improve the effectiveness of mitochondria targeted TBI therapeutics and patient response rates

Cancer treatments significantly elevate OS through multiple mechanisms: chemotherapeutic agents, particularly alkylating agents and platinum compounds, generate excessive ROS directly damaging sperm DNA and membranes (87, 88), while radiation therapy induces both acute and chronic OS states in testicular tissue, leading to prolonged spermatogenic dysfunction (95)