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Description
however, RB1 loss leads to excessive E2F1 activation, resulting in uncontrolled BNIP3 expression and autophagic cell death under hypoxic or nutrient-deprived conditions
Life 12 (11), 1865
The tight junctions of endothelial cells become impaired, facilitating the passage of nanoparticles into the brain and causing damage (24)
Antioxidant properties of UA Several clinical studies have found a relationship between high levels of UA and endothelial damage (the first critical step towards developing atherosclerosis), hypertension, and cardiovascular disease due to the pro-oxidative properties of UA
