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Description
Intestinal microorganisms metabolize TML into trimethyl-5-aminovaleric acid (TMAVA), which can competitively inhibit the binding of -BB to BBD, thereby inhibiting carnitine biosynthesis and FAO in the liver, promoting steatosis and aggravating fatty liver [250]

Microdialysis studies have proven that this specific transporter is functionally responsible for pulling ALCAR from the systemic bloodstream directly into the extracellular fluid of the brain

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