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Targeting Downstream Effectors of ER Activation While ER-targeted therapies are likely to suffer from some of the same risks as estrogen supplementation (115), targeting selected downstream effectors of ER may represent an opportunity to capture the metabolic benefits while bypassing adverse effects associated with estrogens
K.WarholmM.et al (1985)
This supports the position of mtDNA content as a potential predictor of immune recovery success
Excess copper within cells can also increase free radicals, exacerbate DNA damage, thereby promoting the proliferation of tumor cells (146)
