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The above results suggest that VPA down-regulates the expression including Cpt1a, Cpt2 and Lcad, which may cause the accumulation of LCACs
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Additionally, in vitro studies with pituitary cells suggest a similar increase in GH release upon exposure to DSIP, albeit with a notable decline at higher concentrations
Another feature of AD is the accumulation of intracellular neurofibrillary tangles formed by phosphorylated Tau protein (50)
