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However, the accumulated data suggest that the overactivation of this signaling system could stem from inactivation or genetic ablation of the tuberous sclerosis complex (TSC) [261, 262], the master negative regulator of mTOR
Additionally, the mechanism by which the two treatments exert their beneficial effects remains unclear
doi: 10.1016/s0163-7258(03)00052-4
Samir H, Karen A, Ashmawy T, Abo-Ahmed M, El-Sayed M, Watanabe G
