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Interestingly, in traumatic brain injury, GPx1 deficiency was associated with decreased mitochondrial bioenergetic capacity compared to wildtype mice, whereas transgenic GPx1 mice had no decrease in mitochondrial function [114] GPx1 deficiency also enhanced apoptosis and inflammation in a cold-induced brain injury model [115]
AAV2-GAD gene therapy for advanced Parkinsons disease: a double-blind, sham-surgery controlled, randomised trial
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reported that CXCL17 production increased in the lungs of mice infected with M
