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doi: 10.1016/S1474-4422(15)00380-4 59
Many studies have suggested that arrestins compete with G proteins for binding sites in the receptor transmembrane core and sterically preclude further G protein coupling, leading to receptor desensitization [1, 7]
In contrast to myeloid-derived immune cells, keratinocytes express high levels of TLR3 and MAVS, but not TLR4, -7, -8, or -9 [42,68,69]
L., Perevoshchikova, I
