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1993;106(Pt 4):36370
Similarly, hepatic oxalate overproduction due to the loss of AGXT enhances atherosclerosis, while overexpression of AGXT in hepatocytes lowers oxalate and ameliorates atherosclerosis in Apoe / mice 24

Pharmacodynamic monitoring and biomarker-guided selection should verify on-lesion activity while safeguarding marrow and mitochondrial function

Metabolic consequences of iron (Fe)-induced redox dysregulation (FeRD) emerged as a critical pathobiological mechanism in the SARS-CoV2-induced HMRD, the ongoing global health crisis
