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Accordingly, immune cell ferroptosis impacts its immunomodulatory function and ultimately manifests itself in TME as reprogramming tumor progression
This is driven by the likelihood of subchondral bone changes as a consequence of failed repair, particularly if the first attempt involved disruption of the tidemark for marrow recruitment
Simons M, Gordon E, Claesson-Welsh L
The timing and stability of these proteins are tightly controlled by post-translational mechanisms, including ubiquitin-mediated degradation
