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Description
Oxidative stress, in turn, induces cellular impairment and apoptosis by means of bioenergetic failure (via mitochondrial damage), depletion of intracellular antioxidants, biomolecular damage, microtubular damage, activation of ion channels, demyelination, neuroinflammation, and impaired mitophagy [10,11,12,13,14]

For the remaining 60% of people, the infection will become chronic [1]

Cholesterol, inflammation and innate immunity

Contraindications Pregnancy and lactation: individual case reports suggest safety during pregnancy (de Bruyn 2015), however data which mainly include in vivo studies (Xi 2008) are insufficient
