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Therefore, IGF1R with one IGF1 bound is the most optimal state for activation, which partially explains the functional importance of negative cooperativity

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Experimental data show that BPC-157 promotes VEGFR2 signaling and upregulates endothelial nitric oxide synthase (eNOS) through the SrcCaveolin-1 pathway, resulting in improved vasomotor tone and blood flow restoration

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Version 1

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Biomolecules 12 (12), 1731

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