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Description
JNK Depletion of GSH by NAPQI results in increased H 2 O 2 release from mitochondria to cytoplasm, which could oxidize thioredoxin and cause thioredoxin to disassociate from apoptosis signaling-regulating kinase 1 (ASK-1) [41]
This effect is typically dose-dependent and resolves when the peptide is discontinued or cycled off
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doi: 10.1186/s12967-023-04447-2
