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However, -catenins response to various RE design variables and the specific mechanisms remain largely unexplored

We also provide mechanistic evidence that oxidative stress induces cardiomyocyte ferroptosis by promoting lipid peroxidation via GSH depletion and GPX4 inactivation as well as iron overload through Bach1-HO-1 signaling

However, intrinsic apoptosis can be induced by cellular stress, such as oxidative stress, calcium overload or DNA damage and is facilitated by the Bcl-2 proteins

doi: 10.1055/s-2000-8144
