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These findings lead us to hypothesize that the reduction in TGF-1 secretion by ECs, which subsequently affects CFs, may be the primary mechanism underlying the PD-1 inhibitor-induced decrease in cardiac interstitial collagen
iPSC-derived MBN from PD patients further showed higher expression of IL-17 receptors, together with enhanced susceptibility towards death induction by recombinant IL-17 or autologous Th17 cells (73)
Measurements were performed in triplicate
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