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Description
10.31436/jop.v4i2.297

From a mechanistic standpoint, more research is needed to clarify the molecular pathways in human subjects: for example, does amino acid supplementation measurably suppress GCN2 activation or enhance mTOR signaling in patient bone biopsies or blood markers

We demonstrate a reduced number of spinous synapses in homozygous and heterozygous neurons, leading to structurally and functionally altered contacts

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