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J.LutterR.van GoolT.et al
EGCG, as a targeted activating compound of SIRT3, may delay cellular senescence and inflammatory processes induced by senescence (Figure 2)
These changes lead to reduced membrane potential, energy failure, and upregulation of hypertrophic and stress markers like proB-type natriuretic peptide (proBNP), indicating a decline in myocardial function [93,113]
The antiapoptotic effects of different doses of -carotene in chronic ethanol-fed rats
