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Although IGF-IR promotes EMT, it does not necessarily mean that inhibition of IGF-IR is able to reverse this phenotype, due to the compensation by other RTKs
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Depletion of TFG impaired lysosomal CHMP4BmNG recruitment to a similar degree to depletion of the obligate ESCRT-I component TSG101, whereas depleting ALIX did not affect CHMP4BmNG recruitment (Fig
Serious and unexpected side effects may occur
