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Indeed, the clinical literature exhibits heterogeneity in design, dosing, duration, and outcome measures, frequent reliance on parent reported scales without uniform blinding, and underpowered samples that hinder strong inference and guideline development ( in vivo studies explicitly exclude female animals to avoid potential confounding effects related to hormonal fluctuations

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Additional data demonstrate that several other factors, such as ChREBP, LXR, hypoxia-induced factor 1 (HIF1), hepatic nuclear factor 4 (HNF4), and peroxisome proliferator-activated receptor gamma (PPAR-) also have roles in the insulin-dependent induction of GCK (161, 270)

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