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The released thio-glucose was catalyzed by GOx into cytotoxic HS while preserving H2O2, leading to disruption of the mitochondrial electron transport chain and dissipation of the mitochondrial membrane potential

Because Johnsons protocola strict set of dietary rules and daily schedulefinally has its second subject

Under these limiting conditions, we find that glutathione catabolism by CHAC1 supports the mitochondrial cysteine pool to sustain the function of the Fe-S proteins critical to oxidative metabolism

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