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Our analysis of these interventions provides mechanistic insight into the influence of the Fe 2+ -glutathione couple on the occurrence of natural ferroptosis across lifespan, at the cellular and organismal level
Inflammatory stimuli upregulate the expression levels of indoleamine 2,3-dioxygenase, which converts tryptophan to immunosuppressive kynurenine metabolites, thereby suppressing pro-inflammatory responses and promoting regulatory T cell proliferation 11
attributed to its FAD cofactor loss (90)
doi: 10.3390/nu14173501 315
