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[DOI] [Google Scholar] 150.Shahriari M., Zahiri M., Abnous K., Taghdisi S.M., Ramezani M., Alibolandi M
Genetic background, environmental and intraluminal factors, and mucosal immune dysregulation have been suggested to be involved in UC pathogenesis [2]
Subsequent studies confirmed a significant correlation between prolactin rhythm dysregulation and metabolic dysfunction (63)
The study suggests, by comparison with indometacin, two distinct mechanisms behind the observed effectsone being prostaglandin inhibition and the other remaining unidentified
