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The interruption of the activity of these proteins would be responsible for the decrease in antioxidant capacity and the increase in the generation of reactive oxygen species (ROS), interrupting the redox balance in cells and, subsequently, leading to lipid peroxidation and damage to cell membranes (Nogueira et al

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miR-770-5p/XBP1-driven pathway controls ESR1 expression and tamoxifen response in luminal breast cancer

2011;6(8): e23710