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4.3.4 Organelle dysfunction CS also impairs the function of critical organelles central to ferroptosis regulation: (1) Mitochondrial Damage: CS stimulates DRP1-dependent mitochondrial fission, resulting in fragmented, dysfunctional mitochondria that overproduce reactive oxygen species (ROS)

These results provide a proof of concept that an orally delivered small-molecule inhibitor of Lp(a) might be a therapeutic option for patients with high levels of Lp(a) who are at risk of either atherosclerotic cardiovascular disease or calcific aortic valve disease

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