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The parasite genome demonstrates significant instability characterized by aneuploidy, copy number variations (CNVs), and single nucleotide polymorphisms that collectively facilitate rapid adaptation to therapeutic interventions ( Leishmania to develop resistance through various mechanisms including altered drug transport, target modification, and enhanced metabolic detoxification pathways ( Leishmania serves as an evolutionary advantage, allowing rapid selection of resistant populations under drug pressure

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It is the hope that future studies will able to identify genetic risk factors associated with the structuring CD to better understand the pathogenesis of the development of intestinal fibrosis in IBD