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Perkins BA, Lovblom LE, Bril V, Scarr D, Ostrovski I, Orszag A, et al

This study included 81 patients with chronic compression neuropathy of the lumbosacral or cervicobrachial region divided into three homogeneous treatment arms

Patients' health status was quantified using the Chronic Fatigue Questionnaire at four different time points: Before treatment, and 1, 3 and 6 months after the initiation of treatment

As stated above, according to several evidence, acute COVID-19 damage to the CNS may play a central role in the pathogenesis of neuropsychiatric symptoms through several pathophysiological mechanisms: dysregulation of inflammation, astrocytic glial damage, glutamatergic impairment (shift in tryptophan metabolism towards kynurenine pathway, Nf-B, oxidative stress, glutamatergic and excitotoxic damage) and possible glymphatic impairment [15, 46, 82]