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Although homocysteine levels increase with age, a reduction in the cardiovascular risk associated with hyperhomocysteinemia also has been reported in subjects older than 45 years (Reis et al, 1995)
Impaired lysosomal flux or sustained oxidative stress, for instance, can convert autophagy from a protective mechanism into a driver of lysosomal dysfunction, mitochondrial stress and neuronal vulnerability 147,148
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