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7 Molecular alteration and dysregulation pathways in EC: distinction between endometrioid EC and serous EC Endometrial endometrioid carcinomas (EECs) are marked by frequent genetic mutations and pathway dysregulations that drive their development and progression (60)
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In contrast, glutathione S-transferase omega 1 (GSTO1) functions as a pro-pyroptotic licensing factor by catalyzing deSSG of core inflammasome assembly components
