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To validate that the observed phenotype was mediated by the dysregulation of the FTOSLC7A11 axis, a few functional rescue assays were performed

The inhibition of autophagy may allow an imbalance in NRF 2 -mediated metabolism as well as redox regulation, which increases iron deposition and lipid peroxidation, promoting cardiomyocyte ferroptosis and the progression of T1DM cardiomyopathy (79)

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