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doi: 10.1016/j.cmet.2018.04.010 139 TavakoliA.AkhgarjandC.AnsarH.HoujaghaniH.KhormaniA.DjafarianK.et al

Prolonged stimulation of T cells by interferon type I (IFN-I) within the tumor microenvironment results in the downregulation of hydroxyacl-coenzyme A (HADHA), thereby disrupting the fatty acid -oxidation pathway

[9] The function of carnitine as a transmitter of fatty acids through the inner membrane of the mitochondria for use in the process of oxidation, facilitating the reabsorption of acetyl CoA, increasing the production of acetylcholine, increasing the production of phospholipids in cell membrane, and preventing the death of neural cells is some of the proposed mechanisms

As an inhibitor of the E1 kinase, dichloroacetate maintains any residual E1 activity of mutant pyruvate dehydrogenase complex in its maximal active (dephosphorylated) form