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Taken together, we speculate that not only reduced GPx4 expression levels but also loss of GPx4 activity play a crucial role in COPD pathogenesis
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In fact, the isoform found in both rats and mice is missing His-18, and their versions of the protein are immune to the formation of toxic oligomers or fibrils (Asthana et al., 2018)
Sci Rep 9(1):16
