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As early as ten years ago, Domenicali et al (112) found that the structural abnormalities of HSA in hospitalized patients with liver cirrhosis, mainly included cysteinylation of Cys-34 (HSA-CYS), sulfinylation (HSA-SO 2 H), partial truncation of the N/C-terminal (HSA-DA and/or HSA-L), glycosylation (HSA-GLYc), and a combination of these isoforms (Table II)

In addition, the type of substrate has a significant impact on the divergence of microbial communities as it provides sources of nutrients and energy for the micro-organisms to grow and multiply

Blanchet-Bardon C