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Description
Ginsenoside Re can effectively prevent METH-induced mitochondrial dysfunction, oxidative damage, microglial activation, activation of proapoptotic factors, and degeneration of dopaminergic neurons by inhibiting the PKC gene (Shin et al., 2014
Shaking creates shearing forces that will destroy the peptide chains
The LR3 variant includes modifications that reduce binding to IGF-binding proteins, allowing the peptide to remain active on cell receptors for a longer period during experimental procedures
in the process, ME-1 generates NADPH
