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10.1002/jcp.29023 Summary Keywords methylglyoxal, kidney injury, ferroptosis, reactive oxygen species, GSH, selenoprotein Citation Zhang Y, Zhang X, Ren J, Sun H, Yang Z and Wang J (2025) Methylglyoxal induces oxidative stress and ferroptosis of renal tubular epithelial cells in acute and chronic kidney injury mice
Like muscle, normal aging is associated with both quantitative and qualitative changes in bone, including alterations in trabecular architecture, mineralization, protein content and the accumulations of microfractures [356]
Nayak, S
These findings demonstrate that NRF2 activity may serve as both a clinical biomarker as well as an actionable therapeutic target
