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Description
doi: 10.3389/fneur.2021.631210
Stroke, 46:1626-1632
It was discovered in the 1950s as a result of investigations into a self-limited hemolysis that occurred after administration of the antimalarial drug primaquine, most commonly in individuals of African or Mediterranean ethnic origin
While HMGB1 contributes to the development of hepatic steatosis, it may also lead to the activation of Kupffer cells and the exacerbation of inflammation [227]
