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Values of RI of MUA were significantly lower in the LC group compared to the control one on Day 0 and Day 8 (0.480.03 versus 0.720.03 and 0.580.03 versus 0.780.02, respectively)

While genetic, epigenetic, and molecular signaling mechanisms in EC physiology and pathophysiology have been extensively explored, the crucial role of EC metabolism in EC dysfunction and atherogenesis remains largely understudied

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