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In tau transgenic mice we saw improvements in brain structure and function by reducing senescent cell burden by 30% with an intermittent treatment strategy (35), which is consistent with preclinical studies on other models of aging and/or disease (37, 45, 46, 101-103, 108-112)
It also promoted the proliferation of hDPCs in vitro and the elongation in ex vivo -cultured HFs
4,5 The tight gap allows only passive diffusion of lipid-soluble drugs at a molecular weight lower than 400-600 Da
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