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Representative drugs like the highly selective HIF-2 antagonist Belzutifan (MK-6482) [488] inhibit the expression of pro-angiogenic and pro-survival genes, alleviating abnormal metabolic burden and vascular disruption in tumor tissues

Our findings suggest that modulating the iron/FoxO1/hepatokine axis could represent an approach to addressing metabolic dysfunction in MASLD, potentially offering greater specificity than global FoxO1 inhibition

DOI: 10.1038/s41467-020-14629-x Assefa B et al

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