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Cerebral perfusion declines contribute to vascular damage, abnormalities in cerebral structure (58, 59), and the presence of cerebral WMLs (60), ultimately leading to brain dysfunction characterized by impaired processing speed and executive function

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In summary, all these analyses demonstrated a robust and significant causal relationship between 7 circulating inflammatory factors and ADHD, including Eotaxin levels, GDNF, FGF-23, TNF-related activation-induced cytokine levels, Adenosine Deaminase levels, CD40L receptor levels, and Urokinase-type plasminogen activator levels