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Description
Given the association of GSSG with the oxidative protein folding environment in the ER, we examined whether etomoxir could make the ER more oxidizing to proteins in the lumen
ML226 sometimes appeared to increase total OGDH-E2 levels
Curr Opin Neuobiol
When critical repair mechanisms falter and cells bypass apoptotic clearance, they persist and replicate with unresolved lesions [9]
