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TMEM106B homozygous KO ( Tmem106b / ) mice are viable and fertile and exhibit no overt behavioral deficits during developmental and adult stages, but show minor myelination deficits in histopathological analysis [132, 187], consistent with human evidence that a de novo mutation in TMEM106B causes HLD [77]
doi: 10.1067/mlc.2000.108754
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Gynecol Endocrinol 1993, 7: 3341
