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In support of this approach, a 2009 combined cell culture/animal study [150] showed that shear stress in blood vessels keeps Nrf2 in an activated state and as such protects against endothelial dysfunction

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8.6 As this paper focuses on DAMP/DNA-dependent mechanisms shared by HIV-1 and SARS-CoV-2, various further, alternative (but non-exclusive) explanations, including effects of the longer activity of the mRNA or direct effects of blocking the ACE2 receptor, have not been ruled out