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This was related to insulins ability to induce GCLC expression (GCLM was unchanged as in hepatocytes)

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So we're coupling movement of toxins to movement of bio

Physiologic and Genetic Factors Influencing Use or Production of Cysteine, Homocysteine, and Taurine Cyst(e)ine Transport Cys uptake can be diminished and its loss from the plasma increased by defects in cystine transport
