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Based on the analysis of the present research data, it is suggested that L can play a role in the regulation of cell signal transduction pathway through its antioxidant, anti-inflammatory effects as well as the conduction velocity of nerve cells

Previous studies have shown that excess intracellular copper induces cell death by targeting lipid-acylated proteins that regulate the mitochondrial tricarboxylic acid (TCA) cycle, which differs from the known canonical mechanisms of regulated cell death

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Hereditary cancer syndromes like BRCA1/2 mutations, Lynch syndrome, and Li-Fraumeni syndrome create elevated lifetime cancer risk that persists regardless of current cancer status
